#Metabolic #MetabolicSyndrome #metabolicHindi
METABOLIC SYNDROME क्या है ये ? || METABOLIC SYNDROME WHAT IS IT ? || विशेषज्ञ -डॉक्टर वी.के.मिश्रा (गैस्ट्रो एवं लिवर) || The Gastro Liver Hospital Kanpur || हॉस्पिटल में सम्पर्क करने के लिए या डॉक्टर से सलहा लेने के लिए इस नंबर पे बात करे :- 9956653103, 9793555775, 9793555776

Scientific facts with evidence
GASTRO LIVER HOSPITAL :- https://www.youtube.com/channel/UCgsBTy-UmF4OL-60iS5CK5w?view_as=subscriber

Dr. Vinod K.Mishra MBBS, MD, DM (Gastro) is a senior gastroenterologist , hepatologist, and endoscopist. He is an alumini of prestigious post graduate institute of medical education & research, (PGIMER) Chandigarh. He is a busy gastroenterologist performing about 1000 endoscopic procedures per month. He specializes in liver problems and all gastrointestinal diseases. He is trained in endoscopy, colonoscopy, ERCP, and endoscopic ultrasound.He has previously worked at All India Institute of Medical Sciences, New Delhi and Medical College, Jabalpur. He was President of UP chapter of Gastroenterology society.

The GASTRO LIVER HOSPITAL, where he works is a gastroenterology and hepatology super speciality hospital. Hospital has MEDICAL,and SURGICAL wings. It has ICU and plenty of accommodation facilities of different levels. The hospital performs all complicated and advanced surgeries under team of very senior and experienced gastro surgeon . All laparoscopic and open surgeries done .Advanced operations as whipple’s preocedure and oespophageal replacement s are done regularly not to mention of routine surgeries.This is possibily the only centre in north india doing cholecystectomy with intra operative ERCP on regular basis for Gall Bladder stone and CBD stones.The endoscopy unit has following facilities—-

• Endoscopy including multiband imaging.
• Colonoscopy
• ERCP
• Endoscopic ultrasound.
• Capsule endoscopy.
• Hydrogen breath test.
• Urea breath
• Fibroscan for liver diseases.

Currently he is working as
Dr. Vinod K. Mishra MD, DM (Gastro)
Chairman and Chief of Gastroenterology, Liver diseases and Endoscopy services at
The Gastro Liver Hospital
Swaroop Nagar Kanpur (UP), India

For online appointment or consultation
https://www.practo.com/kanpur/doctor/vinod-k-mishra-gastroenterologist

for more information visit our website

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Hyperglycemia or high blood sugar ,cause many of the warning signs of diabetes
• Slow-healing cuts and sores
• More hunger
• Peeing a lot
• Blurry vision
• Heavy thirst
. Fatigue
• Vaginal and skin infections
• Slow-healing cuts and sores
• Numb or tingling feet
Early Signs of Diabetes
Both types of diabetes have some of the same telltale warning signs.
. hunger and fatigue
•Peeing more often and being thirstier
.Dry mouth and itchy skin
.blurred vision
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Diabetic ketoacidosis (one of the hyperglycemic crises), DKA, pathophysiology, causes, clinical presentation (signs and symptoms) and treatment. This video is available for instant download licensing here: https://www.alilamedicalmedia.com/-/galleries/narrated-videos-by-topics/diabetes/-/medias/bda71a7a-4598-4b1d-b298-ed06b3c54238-diabetic-ketoacidosis-dka-narrated-animation
Voice by: Penelope Hammet
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Diabetic ketoacidosis, DKA, is an ACUTE and potentially life-threatening complication of diabetes mellitus. DKA is commonly associated with type 1 but type 2 diabetics are also susceptible. DKA is caused by a critically LOW INSULIN level and is usually triggered when diabetic patients undergo further STRESS, such as infections, inadequate insulin administration, or cardiovascular diseases. It may also occur as the FIRST presentation of diabetes in people who did NOT know they had diabetes and therefore did NOT have insulin treatment.
Glucose is the MAJOR energy source of the body. It comes from digestion of carbohydrates and is carried by the bloodstream to various organs. Insulin is a hormone produced by beta-cells of the pancreas and is responsible for DRIVING glucose INTO cells. When insulin is DEFICIENT, glucose can NOT enter the cells; it stays in the blood, causing HIGH blood sugar levels while the cells are STARVED. In response to this metabolic starvation, the body INcreases the levels of counter-regulatory hormones. These hormones have 2 major effects that are responsible for clinical presentation of DKA:
– First, they produce MORE glucose in an attempt to supply energy to the cells. This is done by breaking down glycogen into glucose, and synthesizing glucose from NON-carbohydrate substrates such as proteins and lipids. However, as the cells CANNOT use glucose, this response ONLY results in MORE sugar in the blood. As blood sugar level EXCEEDS the ability of the kidneys to reabsorb, it overflows into urine, taking water and electrolytes along with it in a process known as OSMOTIC DIURESIS. This results in large volumes of urine, dehydration and excessive thirst.
– Second, they activate lipolysis and fatty acid metabolism for ALTERNATIVE fuel. In the liver, metabolism of fatty acids as an alternative energy source produces KETONE bodies. One of these is acetone, a volatile substance that gives DKA patient’s breath a characteristic SWEET smell. Ketone bodies, unlike fatty acids, can cross the blood-brain barrier and therefore can serve as fuel for the brain during glucose starvation. They are, however, ACIDIC, and when produced in LARGE amounts, overwhelm the buffering capacity of blood plasma, resulting in metabolic ACIDOSIS. As the body tries to reduce blood acidity by EXHALING MORE carbon dioxide, a deep and labored breathing, known as Kussmaul breathing may result. Another compensation mechanism for high acidity MOVES hydrogen ions INTO cells in exchange for potassium. This leads to INcreased potassium levels in the blood; but as potassium is constantly excreted in urine during osmotic diuresis, the overall potassium level in the body is eventually depleted. A blood test MAY indicate too much potassium, or hyperkalemia, but once INSULIN treatment starts, potassium moves BACK into cells and hypokalemia may result instead. For this reason, blood potassium level is monitored throughout treatment and potassium replacement is usually required together with intravenous fluid and insulin as primary treatment for DKA.
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(USMLE topics) What is Gestational Diabetes? Pathology, Risk factors, Complications and Treatments. This video is available for instant download licensing here : https://www.alilamedicalmedia.com/-/galleries/narrated-videos-by-topics/common-ob-gyn-problems/-/medias/257bea34-3735-471b-86d3-d514baa666e8-gestational-diabetes-narrated-animation
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Voice by: Ashley Fleming
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Gestational diabetes is a transient form of diabetes mellitus some women may acquire during pregnancy. Diabetes refers to high levels of blood glucose, commonly known as blood sugar. Glucose is the major energy source of the body. It comes from digestion of carbohydrates and is carried by the bloodstream to the body’s cells. But glucose cannot enter the cells on its own; to do so, it requires assistance from a hormone produced by the pancreas called insulin. Insulin induces the cells to take up glucose, thereby removing it from the blood. Diabetes happens when insulin is either deficient or not used effectively. Without insulin, glucose cannot enter the cells; it stays in the blood, causing high blood sugar levels.
During pregnancy, a temporary organ develops to connect the mother and the fetus, called the placenta. The placenta supplies the fetus with nutrients and oxygen, as well as produces a number of hormones that work to maintain pregnancy. Some of these hormones impair the action of insulin, making it less effective. This insulin-counteracting effect usually begins at about 20 to 24 weeks of pregnancy. The effect intensifies as the placenta grows larger, and becomes most prominent in the last couple of months. Usually, the pancreas is able to adjust by producing more insulin, but in some cases, the amount of placental hormones may become too overwhelming for the pancreas to compensate, and gestational diabetes results.
Any woman can develop gestational diabetes, but those who are overweight or have family or personal history of diabetes or prediabetes are at higher risks. Other risk factors include age, and having previously given birth to large babies.
While gestational diabetes usually resolves on its own after delivery, complications may arise if the condition is severe and/or poorly managed.
Because of the constant high glucose levels in the mother’s blood, the fetus may receive too much nutrients and grow too large, complicating the birth process, and a C-section may be needed for delivery.
High levels of glucose also stimulate the baby’s pancreas to produce more insulin than usual. Shortly after delivery, as the baby continues to have high insulin levels but no longer receives sugar from the mother, the baby’s blood sugar levels can drop suddenly and become exceedingly low, causing seizures. The newborn’s blood sugar level must therefore be monitored and corrected with prompt feeding, or if necessary, with intravenous glucose.
High blood sugar may also increase the mother’s blood pressure and risks of preterm birth. Future diabetes in both mother and child is also more likely to occur.
Gestational diabetes can be successfully managed, or even prevented, with healthy diets, physical exercise, and by keeping a healthy weight before and during pregnancy. In some cases, however, medication or insulin injection may be needed.
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#Metabolicsyndrome #Doctorq #MalayalamNews #KeralaLatestNews #News18

Metabolic syndrome is a cluster of conditions — increased blood pressure, high blood sugar, excess body fat around the waist, and abnormal cholesterol or triglyceride levels — that occur together, increasing your risk of heart disease, stroke and diabetes.

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One evening a young boy, Pâris (9), has a nightly hypo – meaning that his blood sugar drops too low. It is a very stressful and scary experience, both for Pâris and his family. The whole family gets involved and his parents must make the difficult decision on whether to give their son a glucagon injection or not.

This is a short clip from the documentary film Safely in the Rainbow, which is filmed over 16 years. Most of this particular scene was filmed by Pâris’s sister, Antje, who was only 11 years old at the time.

Watch the full film here: https://youtu.be/d-_X-pOKr8k

The film is produced by Wendy and Frédéric Mangeant in collaboration with Novo Nordisk.

You can contact Wendy via info@novonordisk.com
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Almost every person using insulin will have a low blood sugar reaction at some time. Low blood sugar, or hypoglycemia, happens quickly and needs to be treated right away.
With a low blood sugar level, you may have trouble doing simple things. It is important to know the signs of low blood sugar so you can treat it quickly. Your family, friends, teachers and sitters need to know the signs of low blood sugar, too. When they know what to look for, they may notice the signs before you do.
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Your diet plays an important role in managing diabetes. One reason is that the food you eat on a day to day basis has a direct impact on your blood sugar levels. For instance, high carb foods raise your blood sugar levels. The digestive system breaks down the digestible ones into sugar, which enters the blood. But then again, not all carbohydrates are bad. Complex carbohydrates like whole grains take longer to digest while simple carbs like white flour and refined sugar may cause sudden spikes in your blood sugar levels.
Indian foods are deeply rooted in history and are probably one of the oldest foods known to mankind. Traditional Indian foods are extremely diverse, just as its geography. Many staple foods, species and vegetables in the Indian diet are very good for people with diabetes.
So, in this video we will be discussing 5 such traditional Indian foods that have been scientifically proven to have health benefits for people suffering from diabetes.
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This is the seventh video of our diabetes series, “Taking charge of our child’s diabetes.” In this video, you will learn about the two types of diabetic emergencies, severe hypoglycemia, diabetic ketoacidosis. The video answers the following questions: What is severe hypoglycemia? How is a low blood glucose emergency treated? What is diabetic ketoacidosis (DKA)? What causes DKA, what are the common signs and symptoms of DKA, What to do during a diabetic emergency? When to call 911 (Emergency)? In this video, we also demonstrate how to use the glucagon injection.

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Damage to the nervous system is the most common complication of diabetes mellitus. Types of diabetic neuropathy: peripheral neuropathy, autonomic neuropathy, mononeuropathy and proximal neuropathy. Pathophysiology, symptoms, complications and treatments. For patient education. This video is available for instant download licensing here: https://www.alilamedicalmedia.com/-/galleries/narrated-videos-by-topics/diabetes/-/medias/3eea39ab-f6be-4dbd-b7ef-131d90d59b3a-diabetic-neuropathy-narrated-animation
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Voice by : Marty Henne
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All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Diabetic neuropathy is nerve damage caused by diabetes. It is the most common diabetic complication, affecting at least 50% of all patients.
Chronic high blood sugar levels cause progressive injury to neurons. Sensory neurons are usually the first to be affected, followed by autonomic neurons that control internal organs. Rarely, motor neurons that activate voluntary movements may also be impaired later in the disease, albeit to a lesser extent.
There are several types of diabetic neuropathy. A patient may develop more than one type at a time.
The most common is peripheral neuropathy. It’s also called “distal symmetric polyneuropathy” because it typically involves multiple peripheral nerves, on both sides of the body, and affects the longest sensory axons that convey sensations from the feet, legs, and hands. Symptoms develop on the body in that order and include numbness, tingling, or burning sensation, which usually worsen at night. Sensitivity to pain can either be exaggerated, or, on the contrary, lost. Loss of sensation leads to increased risk of painless injuries, which often go unnoticed and therefore untreated. This, together with slow healing caused by diabetes, can turn minor cuts or blisters into serious infections.
Autonomic neuropathy affects nerves that control activities of internal organs.
In cardiac autonomic neuropathy, nerves that regulate heart rate and blood pressure are damaged. As a result, the body reacts more slowly to change of position, and patients may feel lightheadedness when standing up. Other signs include rapid heart rates, or sudden, unexplained changes in heart rate.
In the digestive system, nerve damage may cause nausea, vomiting, loss of appetite, difficulty swallowing, slow stomach emptying, and bowel problems.
Neuropathy in the urinary and reproductive systems may lead to urinary retention, hesitancy, urinary incontinence, and sexual dysfunctions.
Nerve damage in the eyes can cause slower adjustment to changes in light and darkness.
Nerve damage in sweat glands may result in either absence of sweat or heavy sweating, especially at night; different parts of the body may also produce sweat differently.
Another effect of autonomic neuropathy is hypoglycemia unawareness, meaning the body is unaware when blood sugar levels are low. Warning signs such as hunger or dizziness cannot be felt, and patients may pass out before taking steps to increase their blood sugar.
Mononeuropathy, or focal neuropathy, is dysfunction of a single nerve, typically due to entrapment. A common example is carpal tunnel syndrome, in which the median nerve is compressed as it passes through a narrow tunnel in the wrist, causing pain, numbness, and tingling in the hand. Damage to a cranial nerve can cause problems with vision or loss of control of facial muscles.
Proximal neuropathy is a rare type of nerve damage affecting the thighs, hip, buttock, and occasionally, the abdomen or chest, typically on one side of the body. Symptoms include severe pain in the affected areas, and thigh muscle weakness and wasting.
Diabetic neuropathy can be prevented by effectively controlling blood sugar levels. Managing diabetes also helps slow the progression of damage.
Treatments aim to relieve pain and symptoms, to restore functions and prevent further complications. Treatments vary depending on the affected organs.

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Too low in pregnancy is different than the definition of low in a non-pregnant adult. Different phases of pregnancy pose different risks of severe lows, which are more common in early pregnancy. Fingersticks need to be used to validate CGM measured lows. Stacking of insulin needs to be avoided to avoid swinging from high to low and back again.

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In Peter Attia’s podcast The Drive Episode 194, he interviews Rick Johnson and they spend over two hours explaining how fructose causes metabolic disease: obesity, type 2 diabetes, etc. But it needs to be simplified and explained, and that’s what I do in this video!

Dr. Johnson’s hypothesis is that fructose is prime cause of metabolic syndrome and the rapid uptick of modern diseases. He writes about it in his new book “Nature Wants Us to Be Fat”. It explains a lot:

Why it’s harder to stay lean when we get older.
Why athletes seem like they can eat anything they want.
Why most traditional calorie restrictions diets are so hard to maintain
Why keto, low-carb, and paleo diets have had so much success

And importantly, his theory of fructose metabolism is still totally consistent with thermodynamics, and the equation “Calories in minus calories out equals weight gained” is still true.

Link to Podcast episode: https://youtu.be/V02z9mqTWzg
Amazon Affiliate link to Dr. Johnsons most recent book “Nature Wants Us to Be Fat”: https://amzn.to/3BmISvM
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Low blood sugar is a reading of 70 mg or less. Your body gives you signs that your blood sugar is low.
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